Lung Epithelial Injury by B. Anthracis Lethal Toxin Is Caused by MKK-Dependent Loss of Cytoskeletal Integrity
2009

How Bacillus anthracis Lethal Toxin Affects Lung Cells

publication 10 minutes Evidence: moderate

Author Information

Author(s): Lehmann Mandy, Noack Deborah, Wood Malcolm, Perego Marta, Knaus Ulla G., Hartl Dominik

Primary Institution: The Scripps Research Institute

Hypothesis

Lethal toxin from Bacillus anthracis disrupts lung epithelial barrier function through MKK-dependent mechanisms.

Conclusion

The study found that Bacillus anthracis lethal toxin impairs lung epithelial barrier integrity and cell motility, but certain treatments can help restore function.

Supporting Evidence

  • Lethal toxin significantly decreased transepithelial electrical resistance in lung epithelial cells.
  • Long-term exposure to lethal toxin caused a unique cellular phenotype with increased actin filament assembly.
  • Treatment with keratinocyte growth factor improved epithelial barrier integrity in toxin-exposed cells.

Takeaway

Bacillus anthracis toxin makes lung cells weak and unable to heal properly, but some medicines can help them recover.

Methodology

The study used ex vivo differentiated human lung epithelium to assess the effects of lethal toxin on barrier function and cytoskeletal dynamics.

Limitations

The study primarily focused on in vitro models, which may not fully replicate in vivo conditions.

Statistical Information

P-Value

p<0.0001

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.pone.0004755

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