Vesicular Egress of Non-Enveloped Lytic Parvoviruses Depends on Gelsolin Functioning
2008

Gelsolin's Role in Parvovirus Egress

publication Evidence: high

Author Information

Author(s): Bär Séverine, Daeffler Laurent, Rommelaere Jean, Nüesch Jürg P. F.

Primary Institution: Deutsches Krebsforschungszentrum, Heidelberg, Germany

Hypothesis

Does gelsolin play a role in MVM-induced alteration of the actin network and in MVM propagation?

Conclusion

Gelsolin is essential for the efficient egress of progeny virions during MVM infection.

Supporting Evidence

  • Gelsolin was found to control MVM transport from the nucleus to the cell periphery.
  • Gelsolin-dependent actin degradation was essential for progeny virus release.
  • Mutant gelsolins that inhibit gelsolin activity led to reduced release of infectious virions.

Takeaway

Gelsolin helps viruses escape from infected cells by managing the actin network, which is important for their movement.

Methodology

The study involved infecting A9 cells with MVM and analyzing the role of gelsolin in actin modulation and virus propagation through various assays including immunofluorescence and Southern blotting.

Limitations

The study primarily focuses on a specific cell line and may not fully represent the behavior in other cell types or in vivo conditions.

Digital Object Identifier (DOI)

10.1371/journal.ppat.1000126

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