Bone Marrow Suppression by c-Kit Blockade Enhances Tumor Growth of Colorectal Metastases through the Action of Stromal Cell-Derived Factor-1
2012

Bone Marrow Suppression by c-Kit Blockade Enhances Tumor Growth of Colorectal Metastases

Sample size: 16 publication 10 minutes Evidence: moderate

Author Information

Author(s): Rupertus Kathrin, Haberl Gudrun C. Y., Scheuer Claudia, Menger Michael D., Schilling Martin K., Kollmar Otto

Primary Institution: University of Saarland

Hypothesis

Does targeting the migration of hematopoietic cells through c-Kit blockade affect tumor growth and angiogenesis in colorectal metastases?

Conclusion

Bone marrow suppression via anti-c-Kit treatment enhances tumor cell engraftment of colorectal metastases through the SDF-1/CXCR4 pathway.

Supporting Evidence

  • Blockade of c-Kit significantly enhanced tumor cell engraftment compared to controls.
  • Neutralization of SDF-1 completely antagonized the tumor growth associated with c-Kit blockade.
  • C-Kit blockade increased VEGF and CXCR4 expression within the tumors.
  • Histological analysis showed significant tumor cell infiltration in anti-c-Kit treated animals.

Takeaway

When we block a certain protein (c-Kit) that helps blood cells move, tumors can grow faster because the blood cells help them grow. But if we block another protein (SDF-1), it stops the tumors from growing so quickly.

Methodology

BALB/c mice were pretreated with an anti-c-Kit antibody followed by implantation of colorectal cancer cells, with some receiving an anti-SDF-1 antibody.

Potential Biases

Potential bias in the interpretation of results due to the use of a single animal model.

Limitations

The study was conducted in a murine model, which may not fully replicate human tumor biology.

Participant Demographics

Female BALB/c mice, body weight 18–20g.

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1155/2012/196957

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication