Cell killing and DNA damage by etoposide in Chinese hamster V79 monolayers and spheroids: influence of growth kinetics, growth environment and DNA packaging
1993

Etoposide Toxicity in Chinese Hamster V79 Cells

Sample size: 100000 publication 10 minutes Evidence: moderate

Author Information

Author(s): P.L. Olive, J.P. Banath, H.H. Evans

Primary Institution: British Columbia Cancer Research Center

Hypothesis

How does the growth environment and DNA packaging affect the sensitivity of V79 cells to etoposide?

Conclusion

V79 spheroids are more resistant to etoposide than monolayers due to factors like growth fraction and possibly DNA conformation.

Supporting Evidence

  • Cells from V79 spheroids require 50 times more etoposide than monolayers for the same level of cell killing.
  • Outer cells of spheroids are ten times more resistant to etoposide than exponentially growing monolayers.
  • DNA damage measured by the comet assay correlates with cell killing.

Takeaway

When V79 cells grow in clusters called spheroids, they need much more of the drug etoposide to be harmed compared to when they grow in a flat layer.

Methodology

The study involved exposing V79 cells in monolayers and spheroids to etoposide and measuring cell killing and DNA damage using various assays.

Limitations

The study may not account for all factors influencing drug resistance in different cell environments.

Participant Demographics

Chinese hamster V79 lung fibroblasts

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication