Lipofundin-Induced Hyperlipidemia Promotes Oxidative Stress and Atherosclerotic Lesions in New Zealand White Rabbits
2012

Lipofundin-Induced Hyperlipidemia and Atherosclerosis in Rabbits

Sample size: 20 publication 10 minutes Evidence: moderate

Author Information

Author(s): Livan Delgado Roche, Emilio Acosta Medina, Ángela Fraga Pérez, María A. Bécquer Viart, Yosdel Soto López, Viviana Falcón Cama, Ana M. Vázquez López, Gregorio Martínez-Sánchez, Eduardo Fernández-Sánchez

Primary Institution: Center of Studies for Research and Biological Evaluations, University of Havana

Hypothesis

Lipofundin 20% induces hyperlipidemia and oxidative stress, contributing to atherosclerotic lesions in New Zealand white rabbits.

Conclusion

The study found that Lipofundin 20% significantly increases serum lipid levels and promotes oxidative stress, leading to atherosclerotic lesions in rabbits.

Supporting Evidence

  • Lipofundin treatment resulted in significantly increased levels of total cholesterol, triglycerides, LDL, and HDL.
  • Histopathological analysis showed intimal thickening and lipid accumulation in aortic sections from treated rabbits.
  • Oxidative stress markers were significantly elevated in the Lipofundin group compared to controls.

Takeaway

Giving rabbits a fat emulsion called Lipofundin makes their blood fats go up and can cause heart problems.

Methodology

New Zealand rabbits were treated with Lipofundin for 8 days, and various blood and tissue analyses were performed to assess lipid levels and oxidative stress.

Limitations

The study was conducted on a specific animal model, which may not fully represent human atherosclerosis.

Participant Demographics

Standard NZW male rabbits, weighing 2.0–2.5 kg and 12 weeks old.

Statistical Information

P-Value

<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1155/2012/898769

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