Loss of p53 Ser18 and Atm Results in Embryonic Lethality without Cooperation in Tumorigenesis Tumor Analysis of p53S18A; ATM Mutant Mice
2011

Impact of p53 Ser18 and Atm Mutations on Mouse Development and Tumorigenesis

Sample size: 183 publication 10 minutes Evidence: moderate

Author Information

Author(s): Heather L. Armata, Punita Shroff, David E. Garlick, Krista Penta, Andrew R. Tapper, Hayla K. Sluss

Primary Institution: University of Massachusetts Medical School

Hypothesis

The study investigates the role of p53Ser18 in tumor suppression independent of ATM.

Conclusion

The loss of p53Ser18 does not significantly affect survival or tumor distribution in Atm-null mice, but it plays a role in embryonic survival.

Supporting Evidence

  • Loss of p53Ser18 leads to embryonic lethality in compound mutant mice.
  • Atm−/− mice develop tumors predominantly of T-cell origin.
  • p53S18A mice develop lymphomas mostly of B-cell origin.
  • The presence of p53Ser18 mutation does not significantly alter survival in Atm−/− mice.

Takeaway

Mice with certain genetic mutations related to p53 and Atm can have serious health issues, including not surviving before birth, but the exact effects on tumors can vary.

Methodology

The study involved generating and analyzing various mouse strains with specific genetic mutations to assess their survival, tumor development, and growth characteristics.

Potential Biases

Potential bias in the interpretation of tumor types and survival rates due to genetic variability.

Limitations

The study primarily focuses on specific genetic backgrounds and may not generalize to all populations.

Participant Demographics

Mice were on a mixed 129SvEv/C57Bl6 background.

Statistical Information

P-Value

0.007

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.pone.0024813

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