Exhausted CD8 T Cells Downregulate the IL-18 Receptor and Become Unresponsive to Inflammatory Cytokines and Bacterial Co-infections
2011

Exhausted CD8 T Cells and Their Response to Cytokines

Sample size: 16 publication 10 minutes Evidence: high

Author Information

Author(s): Ingram Jennifer T., Yi John S., Zajac Allan J.

Primary Institution: University of Alabama at Birmingham

Hypothesis

Are exhausted CD8 T cells sensitive to activation by combinations of IL-12, IL-18, and IL-21?

Conclusion

Exhausted CD8 T cells lose their ability to respond to certain cytokines, which compromises their function during chronic infections.

Supporting Evidence

  • Exhausted CD8 T cells do not produce IFN-γ in response to antigen-independent stimulation.
  • IL-18Rα expression is downregulated in exhausted CD8 T cells.
  • Memory CD8 T cells retain the ability to respond to cytokine activation.

Takeaway

When the body has a long-lasting virus, some immune cells called CD8 T cells get tired and stop working well. This study shows that these tired cells can't respond to certain signals that usually help them fight infections.

Methodology

The study used LCMV-infected mice to analyze the responses of effector, memory, and exhausted CD8 T cells to various cytokine combinations.

Limitations

The study primarily focused on mouse models, which may not fully replicate human immune responses.

Participant Demographics

Mice of the C57BL/6 strain were used in the experiments.

Statistical Information

P-Value

p<0.001

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.ppat.1002273

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