Genetic Variation in OAS1 Is a Risk Factor for Initial Infection with West Nile Virus in Man
2009

Genetic Variation in OAS1 and West Nile Virus Infection Risk

Sample size: 501 publication 10 minutes Evidence: high

Author Information

Author(s): Lim Jean K., Lisco Andrea, McDermott David H., Huynh Linda, Ward Jerrold M., Johnson Bernard, Johnson Hope, Pape John, Foster Gregory A., Krysztof David, Follmann Dean, Stramer Susan L., Margolis Leonid B., Murphy Philip M.

Primary Institution: National Institutes of Health

Hypothesis

Polymorphisms in the human ortholog of oas1b, OAS1, could influence outcome in humans exposed to West Nile Virus (WNV).

Conclusion

The study identifies OAS1 SNP rs10774671 as a host genetic risk factor for initial infection with West Nile Virus in humans.

Supporting Evidence

  • The frequency of the hypofunctional OAS1 allele is increased in both symptomatic and asymptomatic WNV seroconverters.
  • Individuals homozygous for the 'A' allele showed higher virus accumulation in a model of WNV infection.
  • The AA genotype was significantly greater in WNV-positive subjects than in controls.
  • Statistical analysis showed significant associations across multiple genetic models.

Takeaway

Some people have a gene that makes them more likely to get sick from a virus called West Nile Virus, while others are better at fighting it off.

Methodology

The study analyzed serum or plasma from 501 Caucasian WNV-positive individuals and compared their OAS1 genotypes to healthy controls.

Potential Biases

Potential population stratification could affect the genotypic frequency results.

Limitations

The study is retrospective and limited to North American Caucasians, which may not generalize to other populations.

Participant Demographics

Caucasian individuals from five US centers, including symptomatic and asymptomatic WNV seroconverters.

Statistical Information

P-Value

0.0002

Confidence Interval

95% CI 1.2–2.0

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.ppat.1000321

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