Engagement of Overexpressed Her2 with GEP100 Induces Autonomous Invasive Activities and Provides a Biomarker for Metastases of Lung Adenocarcinoma Tumor Progression with Her2 Overexpression & GEP100
2011

Her2 and GEP100 Interaction in Lung Adenocarcinoma

Sample size: 132 publication 10 minutes Evidence: moderate

Author Information

Author(s): Menju Toshi, Hashimoto Shigeru, Hashimoto Ari, Otsuka Yutaro, Handa Haruka, Ogawa Eiji, Todo Yoshinobu, Wada Hiromi, Date Hiroshi, Sabe Hisataka

Primary Institution: Kyoto University, Hokkaido University

Hypothesis

Does the interaction between overexpressed Her2 and GEP100 contribute to the invasive activities of lung adenocarcinoma cells?

Conclusion

The study found that overexpression of both Her2 and GEP100 in lung adenocarcinoma is correlated with increased invasive activities and node-metastasis.

Supporting Evidence

  • Overexpression of Her2 is linked to poor prognosis in lung cancer.
  • GEP100 expression is heterogeneous within lung adenocarcinoma specimens.
  • Blocking the Her2-GEP100 pathway reduces cancer cell invasion.
  • High levels of GEP100 expression correlate with node-metastasis in lung adenocarcinoma.

Takeaway

When certain cancer cells have too much Her2 and GEP100, they can grow and spread more easily in the body.

Methodology

The study involved analyzing lung adenocarcinoma specimens for Her2 and GEP100 expression and conducting Matrigel invasion assays on H522 cells.

Potential Biases

Potential bias in scoring immunohistochemical staining due to subjective interpretation by investigators.

Limitations

The study's findings may not be generalizable to all lung cancer types due to the focus on adenocarcinoma.

Participant Demographics

Patients with primary lung adenocarcinomas, aged 31-83, with a mix of current, former, and never smokers.

Statistical Information

P-Value

p=0.0197

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.pone.0025301

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