Gastroparesis is associated with oxytocin deficiency, oesophageal dysmotility with hyperCCKemia, and autonomic neuropathy with hypergastrinemia
2009

Oxytocin and GI Issues in Diabetics

Sample size: 19 publication 10 minutes Evidence: moderate

Author Information

Author(s): Borg Julia, Melander Olle, Johansson Linda, Uvnäs-Moberg Kerstin, Rehfeld Jens F, Ohlsson Bodil

Primary Institution: Malmö University Hospital, Lund University

Hypothesis

Do hormone levels differ in diabetics with normal and delayed gastric emptying?

Conclusion

The study found that oxytocin secretion is reduced in patients with delayed gastric emptying, while CCK and gastrin levels are increased in patients with oesophageal dysmotility and autonomic neuropathy.

Supporting Evidence

  • Patients with normal gastric emptying had increased oxytocin levels after a meal.
  • Delayed gastric emptying showed no increase in oxytocin secretion.
  • CCK and gastrin levels increased post-meal in all patients, regardless of gastric emptying status.
  • Patients with oesophageal dysmotility had higher basal CCK levels.
  • Gastrin levels were higher in patients with autonomic neuropathy.

Takeaway

This study looked at how certain hormones behave in diabetics with stomach problems. It found that some hormones don't work as well when the stomach is slow to empty.

Methodology

Nineteen diabetic patients underwent gastric emptying scintigraphy, oesophageal manometry, and blood tests after a fat-rich meal.

Potential Biases

Potential bias due to the exclusion of patients with regular use of medications affecting GI motility.

Limitations

The study had a small sample size and excluded patients with certain conditions, which may limit the generalizability of the findings.

Participant Demographics

19 patients, 10 women, 17 with type 1 diabetes and 2 with type 2 diabetes.

Statistical Information

P-Value

p = 0.015 for oxytocin in normal gastric emptying

Confidence Interval

[116.0–362.5] for delayed gastric emptying

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/1471-230X-9-17

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