Expression of the RAE-1 Family of Stimulatory NK-Cell Ligands Requires Activation of the PI3K Pathway during Viral Infection and Transformation
2011

How a Virus Affects Immune Cell Recognition

publication 10 minutes Evidence: high

Author Information

Author(s): Tokuyama Maria, Lorin Clarisse Delebecque, Frederic Jung, David H. Raulet, Laure Coscoy

Primary Institution: University of California, Berkeley

Hypothesis

Does the activation of the PI3K pathway during viral infection influence the expression of NKG2D ligands?

Conclusion

The study found that activation of the PI3K pathway is essential for the expression of the RAE-1 family of NKG2D ligands during viral infection.

Supporting Evidence

  • RAE-1 expression was significantly induced in MCMV-infected fibroblasts.
  • PI3K pathway activation was necessary for RAE-1 induction.
  • Inhibition of the PI3K pathway blocked RAE-1 surface expression.
  • RAE-1 induction occurred only in infected cells, not in neighboring uninfected cells.
  • Viral gene expression was required for RAE-1 induction.

Takeaway

When a virus infects cells, it can change how those cells are recognized by the immune system. This study shows that a specific pathway in the cell, called the PI3K pathway, is important for this process.

Methodology

The researchers used mouse fibroblasts infected with murine cytomegalovirus (MCMV) to study the expression of RAE-1 ligands and the role of the PI3K pathway.

Limitations

The study primarily focused on mouse models, which may not fully represent human responses.

Participant Demographics

Mouse fibroblasts were used in the experiments.

Statistical Information

P-Value

p<0.01

Statistical Significance

p<0.01

Digital Object Identifier (DOI)

10.1371/journal.ppat.1002265

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