Oncolytic herpes simplex virus propagates tertiary lymphoid structure formation via CXCL10/CXCR3 to boost antitumor immunity
2025

Oncolytic herpes simplex virus boosts antitumor immunity by forming tertiary lymphoid structures

Sample size: 16 publication 10 minutes Evidence: high

Author Information

Author(s): Zhang Meng‐Jie, Lin Wen‐Ping, Wang Qing, Wang Shuo, Song An, Wang Yuan‐Yuan, Li Hao, Sun Zhi‐Jun

Primary Institution: Wuhan University

Hypothesis

The study investigates the role of oncolytic herpes simplex virus (oHSV) in inducing tertiary lymphoid structure (TLS) formation to enhance antitumor immunity.

Conclusion

The findings indicate that oHSV promotes TLS formation and enhances the immune response, improving cancer treatment outcomes.

Supporting Evidence

  • oHSV therapy significantly suppressed tumor growth in both tumor models.
  • TLS formation was observed in 5 out of 16 tumors in the HNSCC model.
  • CXCL10 and CXCR3 were identified as favorable prognostic factors for cancer patients.

Takeaway

This study shows that a special virus can help the body fight cancer better by making new structures that help immune cells work together.

Methodology

The study used mouse models of cancer to assess the effects of oHSV on TLS formation and immune cell infiltration.

Participant Demographics

C57BL/6 female mice, aged 6–8 weeks.

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1111/cpr.13740

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