Mitral Cells of the Olfactory Bulb Perform Metabolic Sensing and Are Disrupted by Obesity at the Level of the Kv1.3 Ion Channel
2011

How Obesity Affects the Olfactory System's Response to Insulin

Sample size: 81 publication 10 minutes Evidence: high

Author Information

Author(s): Fadool Debra Ann, Tucker Kristal, Pedarzani Paola

Primary Institution: The Florida State University

Hypothesis

The olfactory bulb functions as an internal sensor of metabolism via insulin-induced modulation of the potassium channel Kv1.3.

Conclusion

The study found that obesity disrupts the normal insulin response in the olfactory bulb, affecting how mitral cells process sensory information.

Supporting Evidence

  • Insulin application increased firing frequency in mitral cells.
  • Obese mice showed altered action potential firing patterns.
  • Chronic insulin exposure led to resistance in mitral cell response.

Takeaway

This study shows that the part of the brain that helps us smell can also sense how much energy we have, and being overweight can mess that up.

Methodology

The study used adult slice preparations of the olfactory bulb to measure the effects of insulin on mitral cell firing properties.

Potential Biases

Potential bias in the selection of mouse strains and dietary conditions.

Limitations

The study primarily focused on male mice and may not generalize to females or other species.

Participant Demographics

Adult male mice (C57BL6/J strain) were used in the experiments.

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.pone.0024921

Want to read the original?

Access the complete publication on the publisher's website

View Original Publication