DNA double-strand break induction in Ku80-deficient CHO cells following Boron Neutron Capture Reaction
2011

DNA Damage from Boron Neutron Capture Reaction in Ku80-Deficient Cells

publication Evidence: moderate

Author Information

Author(s): Kinashi Yuko, Takahashi Sentaro, Kashino Genro, Okayasu Ryuichi, Masunaga Shinichiro, Suzuki Minoru, Ono Koji

Primary Institution: Research Reactor Institute, Kyoto University

Hypothesis

Boron neutron capture reaction (BNCR) will increase cell killing and slow disappearance of repair protein-related foci to a greater extent in DNA repair-deficient cells than in wild-type cells.

Conclusion

BNCR induces high cytotoxic effects and low capacity to repair DNA double-strand breaks in Ku80-deficient cells.

Supporting Evidence

  • The number of gamma-H2AX foci in xrs-5 cells at 60-120 min after BNCT correlated with the cell killing effect of BNCR.
  • In contrast, foci levels in the xrs-5 cells were significantly higher than in CHO-K1 cells two hours after irradiation.
  • The RBE following BNCR of radio-sensitive mutant cells was lower than that of radio-resistant cells.

Takeaway

This study shows that certain cells that can't repair their DNA are more affected by a special type of radiation treatment, making them more sensitive to it.

Methodology

CHO-K1 and xrs-5 cells were irradiated by thermal neutrons, and DNA double-strand breaks were evaluated by measuring gamma-H2AX and 53BP1 foci using immunofluorescence.

Limitations

The study primarily focuses on two cell lines, which may limit the generalizability of the findings.

Statistical Information

P-Value

p<0.05

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1186/1748-717X-6-106

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