KIR and HLA Loci Are Associated with Hepatocellular Carcinoma Development in Patients with Hepatitis B Virus Infection: A Case-Control Study
2011

KIR and HLA Genes Linked to Liver Cancer in Hepatitis B Patients

Sample size: 333 publication 10 minutes Evidence: moderate

Author Information

Author(s): Pan Ning, Jiang Wei, Sun Hang, Miao Fengqin, Qiu Jie, Jin Hui, Xu Jinhuan, Shi Qian, Xie Wei, Zhang Jianqiong

Primary Institution: Southeast University Medical School, Nanjing, Jiangsu Province, China

Hypothesis

Do KIR and HLA genes influence the risk of hepatocellular carcinoma (HCC) development in patients with hepatitis B virus (HBV) infection?

Conclusion

The study found significant associations between certain KIR and HLA genes and the incidence of HCC in HBV-infected patients, suggesting that NK cell activation may contribute to HCC development.

Supporting Evidence

  • HLA-C1C1 genotype was associated with HCC incidence (p=0.005, OR=2.02).
  • HLA-Bw4-80I increased the risk of HCC development (p=0.0002, OR=2.67).
  • The combination of KIR2DS4 and 1D was linked to disease progression towards HCC (p=0.017, OR=1.89).
  • More risk factors correlated with higher odds ratios for HCC incidence (P trend=7.4E-05).

Takeaway

This study shows that some genes can make people with hepatitis B more likely to get liver cancer, and it helps doctors understand how to prevent it better.

Methodology

A case-control study involving 333 HBV-infected patients, genotyping KIR and HLA loci to assess their association with HCC.

Potential Biases

Potential bias due to the small sample size and strict matching criteria.

Limitations

The study did not analyze KIR allele polymorphisms and lacked information on HBV genotypes.

Participant Demographics

Patients were aged 20 to 65, all members of the Han population, and matched for age and gender.

Statistical Information

P-Value

p=0.005 for HLA-C1C1 association with HCC

Confidence Interval

95% CI: 1.14–3.20 for KIR2DS4/1D

Statistical Significance

p<0.05

Digital Object Identifier (DOI)

10.1371/journal.pone.0025682

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